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Chem. J54, 1104-1108. , B. Aubrey, F. Camer, and N. Haymes (1978a). Environ. Health. Perspect. 23, 177-181. , N. A. J. Selikoff (1978b). Environ. Health Perspect. 23, 315-319. A. J. Selikoff (1982). JAMA, 247, 2112-2116. The Lymphocyte as Target of Toxicity: a Biochemical Approach to Dialkyltin Induced Immunosuppression A. H. PENNINKS and W. SEINEN Working Group Pathology - Toxicology, Pathology and Pharmacology - Toxicology Department, Faculty of Veterinary Sciences, State University of Utrecht, Biltstraat 172, 3572 BP, Utrecht, The Netherlands ABSTRACT Lymphocyte depletion in thymus and thymus-dependent areas in the peripheral lymphoid organs are the most prominent feature of dialkyltin toxicity in rats.

And Starkey, J. R. , Nature 284:622, 1980b. Touraine, J. , Hadden, J. , Hadden, E. , Good, R. , J. Exp. Med. U5:460, 1977. Van Duuren, B. L. , Progr. Exp. Tumor Res. :31, 1969. Vecchi, A. Effect of 2,3,7,8-tetrachlorodibenzo-p-dioxin on macrophase and natural killer cell-mediated cytotoxicty in mice. Biomedicine 180 Dec:32(4):200-4. Warner, N. C. , Woodruff, M. F. A. and Burton, R. C , Int. J. Cancer 20:146, 1977. Weinhold, K. , Goldstein, L. T. and Wheelock, E. , J. Exp. Med. 149:732, 1979. Weinhold, K.

DBTC was the most active but also the other dialkyltins DMTC, DETC and DOTC induced a Pasteur effect in thymocytes; a maximum stimulation of glycolytic activity was observed at concentrations of 120, 10 and 120 μΜ respectively. glucose metabolism of thymocytes c o 3 ■D O > a n O (D T3 c CD ω P +-» o CO "7 CO 3 «0 3* cr. £i o o CO °£ O oo 8| £i o 4. oxygen cons. glucose cons. lactateprod. pyruvate prod. 5. Consumption of glucose and oxygen and the production, of lactate and pyruvate of isolated rat thymocytes incubated with various concentrations of DBTC for 4 hr.

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